| 1. | Foam cells eventually die and further propagate the inflammatory process.
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| 2. | Accumulation of lipid droplets induce the modification of macrophages to foam cells.
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| 3. | ANGPTL4 suppresses foam cell formation to reduce atherosclerosis development.
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| 4. | Macrophages accumulate modified lipid particles and become foam cells.
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| 5. | Low-density lipoprotein ( LDL ) cholesterol is contained by a foam cell.
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| 6. | Instead CXCL5 has a protective role in atherosclerosis by directly controlling macrophage foam cell formation.
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| 7. | Foam cells form the fatty streaks of the plaques of atheroma in the tunica intima of arteries.
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| 8. | Lysis of foam cells resulted in Atherosclerotic plaques and such plaques rupture and blocked the thrombotic vessel.
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| 9. | These LDL molecules are oxidized and taken up by macrophages, which become engorged and form foam cells.
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| 10. | These foam cells often become trapped in the walls of blood vessels and contribute to atherosclerotic plaque formation.
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